Topics of this game:
- The mental action or process of acquiring knowledge and understanding through thought, experience, and the senses
- Decrease in Physical Strength, Endurance, and Flexibility, Decline in Efficiency of Body Organs, Loss of Bone Mass, Slower Reflexes, Sensory Decline
- Secondary to changes in the brain, Slower processing time, Longer learning time, Mild memory reductions, Often improved vocabulary, general knowledge
- Known benefit from maintenance of social interaction, Greater need for social supports, Bereavement/grief
- Possible memory loss, patient more concerned, indept., convo not impaired, anomia, stay social, doesn't get lost
- family more concerned, convo impaired, anomia, dependent, gets lost, loss of social interest
- Rapid onset, identifiable date, alterations in consciousness; reversible
- Complaints of memory loss, fluctuating cognitive loss, mood affected
- Insidious onset, lack of awareness of memory loss, no alterations in consciousness
- Brain disease characterized by neurofibrillary tangles and amyloid plaques
- Proteins between nerve cells (neurons) in the brain
- Protein fibers inside the brain's cells
- CT looks for morphological/structural changes, used in AD to rule out focal lesions, PET scans look at tissue metabolism
- Some/few/all; wandering/pacing/repetitive motor activities; anxiety/agitation/resistant to care, suspicious/delusional/hallucin.; abnormal sleep/wake
- Locus is more anterior, fewer plaques but more excessive tau protein tangles
- Type of FFD, primary deficit is linguistic, may begin in 40s
- AKA CTE
- CBD; greater concentrations of abnormal protein tau. Resembles Parkinson’s but does not respond to Parkinson meds
- CJD; like mad cow; prion protein mutation; spongiform encephalopathies
- vCJD; rare and in younger ppl; may result from human consumption of beef from cattle with bovine spongiform encephalopathy (BSE)
- Young onset (30--40s); familial; personality changes, irritability, depression, psychotic behavior, chorea, weakness, gait disturbances
- Abnormal protein (alpha-synuclein); Lewy Bodies are abnormal structures in the neurons in the substantia nigra. They may also appear in the cortex
